VISUAL VERBAL AGNOSIA
\vˈɪʒuːə͡l vˈɜːbə͡l aɡnˈə͡ʊzi͡ə], \vˈɪʒuːəl vˈɜːbəl aɡnˈəʊziə], \v_ˈɪ_ʒ_uː_əl v_ˈɜː_b_əl a_ɡ_n_ˈəʊ_z_iə]\
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Loss of the power to comprehend written materials despite preservation of the ability to write (i.e., alexia without agraphia). This condition is generally attributed to lesions that "disconnect" the visual cortex of the non-dominant hemisphere from language centers in the dominant hemisphere. This may occur when a dominant visual cortex injury is combined with underlying white matter lesions that involve crossing fibers from the occipital lobe of the opposite hemisphere. (From Adams et al., Principles of Neurology, 6th ed, p483)
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Proto Oncogene Proteins c erbB 2
- cell surface protein-tyrosine kinase that is found to be overexpressed in significant number adenocarcinomas. It has extensive homology can heterodimerize EGF EPIDERMAL GROWTH FACTOR), 3 receptor (RECEPTOR, 3) and the 4 receptor. Activation of erbB-2 receptor occurs during heterodimer formation with a ligand-bound erbB family members. EC 2.7.11.-.