PROTOZOAL MENINGOENCEPHALITIS
\pɹˈə͡ʊtə͡ʊzˌə͡ʊl mˌɛnɪŋɡˌə͡ʊnsɪfɐlˈa͡ɪtɪs], \pɹˈəʊtəʊzˌəʊl mˌɛnɪŋɡˌəʊnsɪfɐlˈaɪtɪs], \p_ɹ_ˈəʊ_t_əʊ_z_ˌəʊ_l m_ˌɛ_n_ɪ_ŋ_ɡ_ˌəʊ_n_s_ɪ_f_ɐ_l_ˈaɪ_t_ɪ_s]\
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Infections of the brain, spinal cord, or meninges by single celled organisms of the subkingdom PROTOZOA. The central nervous system may be the primary or secondary site of protozoal infection. Examples of primary infections include cerebral amebiasis, granulomatous amebic encephalitis, primary amebic meningoencephalitis, and TRYPANOSOMIASIS, AFRICAN. Cerebral malaria, cerebral babesiosis, and chagasic meningoencephalitis are examples of secondary infections. These diseases may occur as OPPORTUNISTIC INFECTIONS or arise in immunocompetent hosts. (From Joynt, Clinical Neurology, 1998, Ch27, pp37-47)
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Proto Oncogene Proteins c erbB 2
- cell surface protein-tyrosine kinase that is found to be overexpressed in significant number adenocarcinomas. It has extensive homology can heterodimerize EGF EPIDERMAL GROWTH FACTOR), 3 receptor (RECEPTOR, 3) and the 4 receptor. Activation of erbB-2 receptor occurs during heterodimer formation with a ligand-bound erbB family members. EC 2.7.11.-.