POSTENCEPHALITIC PARKINSONISM
\pˌə͡ʊstənsˌɛfɐlˈɪtɪk pɑːkˈɪnsənˌɪzəm], \pˌəʊstənsˌɛfɐlˈɪtɪk pɑːkˈɪnsənˌɪzəm], \p_ˌəʊ_s_t_ə_n_s_ˌɛ_f_ɐ_l_ˈɪ_t_ɪ_k p_ɑː_k_ˈɪ_n_s_ə_n_ˌɪ_z_ə_m]\
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Parkinsonism following encephalitis, historically seen as a sequella of encephalitis lethargica (Von Economo Encephalitis). The early age of onset, the rapid progression of symptoms followed by stabilization, and the presence of a variety of other neurological disorders (e.g., sociopathic behavior; TICS, MUSCLE SPASMS; oculogyric crises; hyperphagia; and bizarre movements) distinguish this condition from primary PARKINSON DISEASE. Pathologic features include neuronal loss and gliosis concentrated in the MESENCEPHALON; SUBTHALAMUS; and HYPOTHALAMUS. (From Adams et al., Principles of Neurology, 6th ed, p754)
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Word of the day
Quinones
- Hydrocarbon rings which contain two moieties position. They can be substituted in any position except at the ketone groups.