EARLY POST TRAUMATIC SEIZURE
\ˈɜːlɪ pˈə͡ʊst tɹɔːmˈatɪk sˈiːʒə], \ˈɜːlɪ pˈəʊst tɹɔːmˈatɪk sˈiːʒə], \ˈɜː_l_ɪ p_ˈəʊ_s_t t_ɹ_ɔː_m_ˈa_t_ɪ_k s_ˈiː_ʒ_ə]\
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Recurrent seizures causally related to CRANIOCEREBRAL TRAUMA. Seizure onset may be immediate but is typically delayed for several days after the injury and may not occur for up to two years. The majority of seizures have a focal onset that correlates clinically with the site of brain injury. Cerebral cortex injuries caused by a penetrating foreign object (CRANIOCEREBRAL TRAUMA, PENETRATING) are more likely than closed head injuries (HEAD INJURIES, CLOSED) to be associated with epilepsy. Concussive convulsions are nonepileptic phenomena that occur immediately after head injury and are characterized by tonic and clonic movements. (From Rev Neurol 1998 Feb;26 (150):256-261; Sports Med 1998 Feb;25 (2):131-6)
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Proto Oncogene Proteins c erbB 2
- cell surface protein-tyrosine kinase that is found to be overexpressed in significant number adenocarcinomas. It has extensive homology can heterodimerize EGF EPIDERMAL GROWTH FACTOR), 3 receptor (RECEPTOR, 3) and the 4 receptor. Activation of erbB-2 receptor occurs during heterodimer formation with a ligand-bound erbB family members. EC 2.7.11.-.