ALCOHOLIC AXONAL NEUROPATHIES
\ˌalkəhˈɒlɪk ˈaksɒnə͡l njuːɹˈɒpəθɪz], \ˌalkəhˈɒlɪk ˈaksɒnəl njuːɹˈɒpəθɪz], \ˌa_l_k_ə_h_ˈɒ_l_ɪ_k ˈa_k_s_ɒ_n_əl n_j_uː_ɹ_ˈɒ_p_ə_θ_ɪ_z]\
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A condition where damage to the peripheral nervous system (including the peripheral elements of the autonomic nervous system) is associated with chronic ingestion of alcoholic beverages. The disorder may be caused by a direct effect of alcohol, an associated nutritional deficiency, or a combination of factors. Clinical manifestations include variable degrees of weakness; ATROPHY; PARESTHESIAS; pain; loss of reflexes; sensory loss; diaphoresis; and postural hypotension. (From Arch Neurol 1995;52 (1):45-51; Adams et al., Principles of Neurology, 6th ed, p1146)
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Proto Oncogene Proteins c erbB 2
- cell surface protein-tyrosine kinase that is found to be overexpressed in significant number adenocarcinomas. It has extensive homology can heterodimerize EGF EPIDERMAL GROWTH FACTOR), 3 receptor (RECEPTOR, 3) and the 4 receptor. Activation of erbB-2 receptor occurs during heterodimer formation with a ligand-bound erbB family members. EC 2.7.11.-.